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Artigo de periodico
Increased atrial β-adrenergic receptors and GRK-2 gene expression can play a fundamental role in heart failure after repair of congenital heart disease with cardiopulmonary bypass (2017)
Oliveira, Marcela Silva; Carmona, Fabio; Vicente, Walter Villela de Andrade; Manso, Paulo Henrique; Mata, Karina Magalhães Alves da; Celes, Mara Rúbia Nunes; Campos, Erica C.; Ramos, Simone Gusmão
Source: Pediatric Cardiology. Unidade: FMRP
Subjects: EXPRESSÃO GÊNICA, BIÓPSIA, INSUFICIÊNCIA CARDÍACA, BEBÊS, MIOCÁRDIO, BIOMARCADORES
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ABNT
OLIVEIRA, Marcela Silva et al. Increased atrial β-adrenergic receptors and GRK-2 gene expression can play a fundamental role in heart failure after repair of congenital heart disease with cardiopulmonary bypass. Pediatric Cardiology, v. 38, n. 4, p. 734-745, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00246-017-1573-1. Acesso em: 20 maio 2024.
APA
Oliveira, M. S., Carmona, F., Vicente, W. V. de A., Manso, P. H., Mata, K. M. A. da, Celes, M. R. N., et al. (2017). Increased atrial β-adrenergic receptors and GRK-2 gene expression can play a fundamental role in heart failure after repair of congenital heart disease with cardiopulmonary bypass. Pediatric Cardiology, 38( 4), 734-745. doi:10.1007/s00246-017-1573-1
NLM
Oliveira MS, Carmona F, Vicente WV de A, Manso PH, Mata KMA da, Celes MRN, Campos EC, Ramos SG. Increased atrial β-adrenergic receptors and GRK-2 gene expression can play a fundamental role in heart failure after repair of congenital heart disease with cardiopulmonary bypass [Internet]. Pediatric Cardiology. 2017 ; 38( 4): 734-745.[citado 2024 maio 20 ] Available from: https://doi.org/10.1007/s00246-017-1573-1
Vancouver
Oliveira MS, Carmona F, Vicente WV de A, Manso PH, Mata KMA da, Celes MRN, Campos EC, Ramos SG. Increased atrial β-adrenergic receptors and GRK-2 gene expression can play a fundamental role in heart failure after repair of congenital heart disease with cardiopulmonary bypass [Internet]. Pediatric Cardiology. 2017 ; 38( 4): 734-745.[citado 2024 maio 20 ] Available from: https://doi.org/10.1007/s00246-017-1573-1
Artigo de periodico
Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis (2013)
Celes, Mara R. N.; Malvestio, Lygia M.; Suadicani, Sylvia O.; Prado, Cibele Maria; Figueiredo, Maria J.; Campos, Erica C.; Freitas, Ana C. S.; Spray, David C.; Tanowitz, Herbert B.; Silva, João Santana da; Rossi, Marcos Antonio
Source: PLOS ONE. Unidade: FMRP
Subjects: CARDIOPATIAS, SEPSE, EXPERIMENTOS ANIMAIS
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ABNT
CELES, Mara R. N. et al. Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis. PLOS ONE, v. 8, n. 7, p. e68809-1-e68809-13, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0068809. Acesso em: 20 maio 2024.
APA
Celes, M. R. N., Malvestio, L. M., Suadicani, S. O., Prado, C. M., Figueiredo, M. J., Campos, E. C., et al. (2013). Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis. PLOS ONE, 8( 7), e68809-1-e68809-13. doi:10.1371/journal.pone.0068809
NLM
Celes MRN, Malvestio LM, Suadicani SO, Prado CM, Figueiredo MJ, Campos EC, Freitas ACS, Spray DC, Tanowitz HB, Silva JS da, Rossi MA. Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis [Internet]. PLOS ONE. 2013 ; 8( 7): e68809-1-e68809-13.[citado 2024 maio 20 ] Available from: https://doi.org/10.1371/journal.pone.0068809
Vancouver
Celes MRN, Malvestio LM, Suadicani SO, Prado CM, Figueiredo MJ, Campos EC, Freitas ACS, Spray DC, Tanowitz HB, Silva JS da, Rossi MA. Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis [Internet]. PLOS ONE. 2013 ; 8( 7): e68809-1-e68809-13.[citado 2024 maio 20 ] Available from: https://doi.org/10.1371/journal.pone.0068809
Artigo de periodico
Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy (2012)
Prado, Cibele Maria; Celes, Mara R. N.; Malvestio, Lygia M.; Campos, Erica C.; Silva, João Santana da; Jelicks, Linda; Tanowitz, Herbert B; Rossi, Marco Antônio
Source: Microbes and Infection. Unidade: FMRP
Subjects: DOENÇA DE CHAGAS, IMAGEM POR RESSONÂNCIA MAGNÉTICA, MIOCARDITE (PARASITOLOGIA), MIOCÁRDIO (PATOLOGIA), TRYPANOSOMA CRUZI
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ABNT
PRADO, Cibele Maria et al. Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy. Microbes and Infection, v. 14, n. 1 p. 59-68, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2011.08.010. Acesso em: 20 maio 2024.
APA
Prado, C. M., Celes, M. R. N., Malvestio, L. M., Campos, E. C., Silva, J. S. da, Jelicks, L., et al. (2012). Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy. Microbes and Infection, 14( 1 p. 59-68). doi:10.1016/j.micinf.2011.08.010
NLM
Prado CM, Celes MRN, Malvestio LM, Campos EC, Silva JS da, Jelicks L, Tanowitz HB, Rossi MA. Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy [Internet]. Microbes and Infection. 2012 ; 14( 1 p. 59-68):[citado 2024 maio 20 ] Available from: https://doi.org/10.1016/j.micinf.2011.08.010
Vancouver
Prado CM, Celes MRN, Malvestio LM, Campos EC, Silva JS da, Jelicks L, Tanowitz HB, Rossi MA. Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy [Internet]. Microbes and Infection. 2012 ; 14( 1 p. 59-68):[citado 2024 maio 20 ] Available from: https://doi.org/10.1016/j.micinf.2011.08.010
Artigo de periodico
Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy (2011)
Campos, Erica C.; O'Connell, João L.; Malvestio, Lygia M.; Romano, Minna M. Dias; Ramos, Simone Gusmão; Celes, Mara Rúbia N.; Prado, Cibele M.; Simões, Marcus Vinicius; Rossi, Marcos Antonio
Source: European Journal of Pharmacology. Unidade: FMRP
Subjects: CARDIOPATIAS, INSUFICIÊNCIA CARDÍACA, ANTINEOPLÁSICOS, NEOPLASIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAMPOS, Erica C. et al. Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy. European Journal of Pharmacology, v. 670, n. 2/3, p. 541-553, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2011.09.021. Acesso em: 20 maio 2024.
APA
Campos, E. C., O'Connell, J. L., Malvestio, L. M., Romano, M. M. D., Ramos, S. G., Celes, M. R. N., et al. (2011). Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy. European Journal of Pharmacology, 670( 2/3), 541-553. doi:10.1016/j.ejphar.2011.09.021
NLM
Campos EC, O'Connell JL, Malvestio LM, Romano MMD, Ramos SG, Celes MRN, Prado CM, Simões MV, Rossi MA. Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy [Internet]. European Journal of Pharmacology. 2011 ; 670( 2/3): 541-553.[citado 2024 maio 20 ] Available from: https://doi.org/10.1016/j.ejphar.2011.09.021
Vancouver
Campos EC, O'Connell JL, Malvestio LM, Romano MMD, Ramos SG, Celes MRN, Prado CM, Simões MV, Rossi MA. Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy [Internet]. European Journal of Pharmacology. 2011 ; 670( 2/3): 541-553.[citado 2024 maio 20 ] Available from: https://doi.org/10.1016/j.ejphar.2011.09.021
Artigo de periodico
Coronary microvascular disease in chronic chagas cardiomyopathy including an overview on history, pathology, and other proposed pathogenic mechanisms (2010)
Rossi, Marcos Antonio; Tanowitz, Herbert B.; Malvestio, Lygia M.; Celes, Mara R.; Campos, Erica C.; Blefari, Valdecir; Prado, Cibele M.
Source: PLoS Neglected Tropical Diseases. Unidade: FMRP
Assunto: DOENÇA DE CHAGAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROSSI, Marcos Antonio et al. Coronary microvascular disease in chronic chagas cardiomyopathy including an overview on history, pathology, and other proposed pathogenic mechanisms. PLoS Neglected Tropical Diseases, v. 4, n. 8, p. e674-1 - e674-12, 2010Tradução . . Disponível em: https://repositorio.usp.br/directbitstream/8fe158b2-1dec-4db3-bfa3-ab898653b846/002178829.pdf. Acesso em: 20 maio 2024.
APA
Rossi, M. A., Tanowitz, H. B., Malvestio, L. M., Celes, M. R., Campos, E. C., Blefari, V., & Prado, C. M. (2010). Coronary microvascular disease in chronic chagas cardiomyopathy including an overview on history, pathology, and other proposed pathogenic mechanisms. PLoS Neglected Tropical Diseases, 4( 8), e674-1 - e674-12. Recuperado de https://repositorio.usp.br/directbitstream/8fe158b2-1dec-4db3-bfa3-ab898653b846/002178829.pdf
NLM
Rossi MA, Tanowitz HB, Malvestio LM, Celes MR, Campos EC, Blefari V, Prado CM. Coronary microvascular disease in chronic chagas cardiomyopathy including an overview on history, pathology, and other proposed pathogenic mechanisms [Internet]. PLoS Neglected Tropical Diseases. 2010 ; 4( 8): e674-1 - e674-12.[citado 2024 maio 20 ] Available from: https://repositorio.usp.br/directbitstream/8fe158b2-1dec-4db3-bfa3-ab898653b846/002178829.pdf
Vancouver
Rossi MA, Tanowitz HB, Malvestio LM, Celes MR, Campos EC, Blefari V, Prado CM. Coronary microvascular disease in chronic chagas cardiomyopathy including an overview on history, pathology, and other proposed pathogenic mechanisms [Internet]. PLoS Neglected Tropical Diseases. 2010 ; 4( 8): e674-1 - e674-12.[citado 2024 maio 20 ] Available from: https://repositorio.usp.br/directbitstream/8fe158b2-1dec-4db3-bfa3-ab898653b846/002178829.pdf
Artigo de periodico
Isoproterenol induces primary loss of dystrophin in rat hearts: correlation with myocardial injury (2008)
Campos, Erica C.; Romano, Minna M. D.; Prado, Cibele M.; Rossi, Marcos Antonio
Source: International Journal of Experimental Pathology. Unidade: FMRP
Assunto: MIOCARDIOPATIAS
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ABNT
CAMPOS, Erica C. et al. Isoproterenol induces primary loss of dystrophin in rat hearts: correlation with myocardial injury. International Journal of Experimental Pathology, v. 89, n. 5, p. 367-381, 2008Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2613.2008.00604.x. Acesso em: 20 maio 2024.
APA
Campos, E. C., Romano, M. M. D., Prado, C. M., & Rossi, M. A. (2008). Isoproterenol induces primary loss of dystrophin in rat hearts: correlation with myocardial injury. International Journal of Experimental Pathology, 89( 5), 367-381. doi:10.1111/j.1365-2613.2008.00604.x
NLM
Campos EC, Romano MMD, Prado CM, Rossi MA. Isoproterenol induces primary loss of dystrophin in rat hearts: correlation with myocardial injury [Internet]. International Journal of Experimental Pathology. 2008 ; 89( 5): 367-381.[citado 2024 maio 20 ] Available from: https://doi.org/10.1111/j.1365-2613.2008.00604.x
Vancouver
Campos EC, Romano MMD, Prado CM, Rossi MA. Isoproterenol induces primary loss of dystrophin in rat hearts: correlation with myocardial injury [Internet]. International Journal of Experimental Pathology. 2008 ; 89( 5): 367-381.[citado 2024 maio 20 ] Available from: https://doi.org/10.1111/j.1365-2613.2008.00604.x

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